Patient/caregiver was educated on pathophysiology of left-sided heart failure as follows:

  1. Left side of the heart receives oxygen rich blood from the lungs and pumps this blood to the vital organs of the body through the general circulation.
  2. Strong contractions of the chambers on the left heart are needed for effective pumping of oxygen rich blood to the tissues.
  3. Damaged muscle on left side of heart, secondary to various factors, cannot contract effectively, contributing to poor contractions of the heart chambers.
  4. Ineffective and poor contractions of left side of heart result in inability of heart chambers to pump adequate blood to vital organs and tissues, such as, heart, brain, and kidneys.
  5. Inadequate blood supply to vital organs, in due course of time, contribute to progressively compromised vital organ function.
  6. Inadequate pumping of blood out of heart contributes to increased retention and back-up of blood into the heart chambers on the left side.
  7. Progressive back-up of blood in the heart chambers can increase the pressure within the left side of heart.
  8. This increased pressure in the left-sided chambers resists filling of these chambers with blood coming from the lungs, as the chambers are already loaded with backed up blood.
  9. This eventually results in progressive back-up of blood into the lungs and result in symptoms such as, fluid retention in the chest leading to difficulty in gas exchange in the lungs, poor oxygen pick-up in the lungs, exacerbation of shortness of breath, and compromised endurance.
  10. If this situation is not controlled and the damage continues further, progressive back-up of blood in the chest leads to increased pressures on right side of heart, right-sided heart failure, swelling of the extremities with pitting edema, and acute weight gain.